Tuesday, July 3 2012 16:30-18:30 @ Pavilion theatre
Concurrent session 2.3: Time perception and attention
Ian Phillips, "Attention and The Passing Of Time."
Jan Almang, "Time Consciousness and Object Constancy."
Bence Nanay, "Unconscious Attention."
Sundeep Teki, "A Unified Neuroanatomical Model Of Time Perception."
Gethin Hughes, "The Role Of Action-Effect Prediction In Intentional Binding And Sensory Attenuation."
Nathan Faivre, "Quantative Evaluation Of Conscious And Nonconscious Temporal Integration."
Showing posts with label July-3. Show all posts
Showing posts with label July-3. Show all posts
Tuesday, July 3, 2012
Concurrent sessions 2.2: Prediction, expectation, and consciousness
Tuesday, July 3 2012 16:30-18:30 @ Corn Exchange
Concurrent session 2.2: Prediction, expectation, and consciousness
Lars Muckli, "Predictive Coding In The Visual Cortex."
Maren Urner, "Pre-Stimulus Activity Predicts Awareness In Visual Extinction."
Anil Seth, "An Interoceptive Predictive Coding Model Of Conscious Presence"
Andreas Lind, "Can We Tell What We Said When We Hear Ourselves Saying Something Else?"
Ron Chrisley, "Making Predictive Coding More Predictive, More Enactive."
Michael Madary, "The Anticipation/Fulfilment Model Of Vision Connects Phenomenology And Cognitive Neuroscience."
Concurrent session 2.2: Prediction, expectation, and consciousness
Lars Muckli, "Predictive Coding In The Visual Cortex."
Maren Urner, "Pre-Stimulus Activity Predicts Awareness In Visual Extinction."
Anil Seth, "An Interoceptive Predictive Coding Model Of Conscious Presence"
Andreas Lind, "Can We Tell What We Said When We Hear Ourselves Saying Something Else?"
Ron Chrisley, "Making Predictive Coding More Predictive, More Enactive."
Michael Madary, "The Anticipation/Fulfilment Model Of Vision Connects Phenomenology And Cognitive Neuroscience."
Concurrent sessions 2.1: Implicit learning and perception
Tuesday, July 3 2012 16:30-18:30 @ Dome Theatre
Concurrent session 2.1: Implicit learning and perception
Sid Kouider, "The ‘Sublink’ Effect: Inducing An Attention Blink From Subliminal Stimuli."
Eva Van den Bussche, "The Evolution Of Masked Priming Effects Using The Incremental Priming Technique."
Anne Atas, "Subliminal Sequence Learning In Peripheral Vision."
Qiufang Fu, "Cultural Differences In Implicit Sequence Learning."
Claire Sergent, "Retro-Attention: Triggering Conscious Perception After The Stimulus Is Gone."
Ryota Kanai, "Stimulus Size Has Opposite Impacts On The Speed Of Unconscious Processing And The Timing Of Conscious Perception."
Concurrent session 2.1: Implicit learning and perception
Sid Kouider, "The ‘Sublink’ Effect: Inducing An Attention Blink From Subliminal Stimuli."
Eva Van den Bussche, "The Evolution Of Masked Priming Effects Using The Incremental Priming Technique."
Anne Atas, "Subliminal Sequence Learning In Peripheral Vision."
Qiufang Fu, "Cultural Differences In Implicit Sequence Learning."
Claire Sergent, "Retro-Attention: Triggering Conscious Perception After The Stimulus Is Gone."
Ryota Kanai, "Stimulus Size Has Opposite Impacts On The Speed Of Unconscious Processing And The Timing Of Conscious Perception."
Concurrent sessions 1.3: Unity and the unconscious
Tuesday, July 3 2012 14:00-16:00 @ Pavilion Theatre
Concurrent session 1.3: Unity and the unconscious
Annelinde Vandenbroucke, "Visual Sensory Memory Contains Phenomenal Rather Than Unconscious Representations."
Axel Cleeremans, "Behavioural Priming: It’s All In The Mind, But Whose Mind?"
Philip Pärnaments, "Objective Markers Of Detection Process During A Choice Blindness Task."
Heiko Reuss, "Adaptation To Unconscious Conflicts In Unconscious Contexts."
Ting-An Lin, "Do Split-Brain Subjects Have Unified Consciousness?"
Michael Klincewicz, "Access And The Unity Of Consciousness."
Concurrent session 1.3: Unity and the unconscious
Annelinde Vandenbroucke, "Visual Sensory Memory Contains Phenomenal Rather Than Unconscious Representations."
Axel Cleeremans, "Behavioural Priming: It’s All In The Mind, But Whose Mind?"
Philip Pärnaments, "Objective Markers Of Detection Process During A Choice Blindness Task."
Heiko Reuss, "Adaptation To Unconscious Conflicts In Unconscious Contexts."
Ting-An Lin, "Do Split-Brain Subjects Have Unified Consciousness?"
Michael Klincewicz, "Access And The Unity Of Consciousness."
Concurrent sessions 1.2: Altered states
Tuesday, July 3 2012 14:00-16:00 @ Corn Exchange
Concurrent session 1.2: Altered states
Carolyn Jennings, "Immersion Consciousness"
Adam Shriver, "Is Asymbolia The Only “Genuine” Case Of Dissociation Between The Affective And Sensory Dimensions Of Pain?"
Damian Cruse, "Communicating With The Unconscious: The Development Of A Brain-Computer Interface For The Vegetative And Minimally Conscious States."
Ithabi Gantner, "Thalamic Generator For Propofol-Induced Alpha-Rhythm: A Simultaneous EEG-fMRI Study."
Jacobo Sitt, "Cognitive Capacity But Not Sedation Level Predicts Neural Signatures Of Conscious Processing."
Robin Carhart-Harris, "The Neural Correlates Of Psychedelic Consciousness As Determined By fMRI Studies With Psilocybin."
Concurrent session 1.2: Altered states
Carolyn Jennings, "Immersion Consciousness"
Adam Shriver, "Is Asymbolia The Only “Genuine” Case Of Dissociation Between The Affective And Sensory Dimensions Of Pain?"
Damian Cruse, "Communicating With The Unconscious: The Development Of A Brain-Computer Interface For The Vegetative And Minimally Conscious States."
Ithabi Gantner, "Thalamic Generator For Propofol-Induced Alpha-Rhythm: A Simultaneous EEG-fMRI Study."
Jacobo Sitt, "Cognitive Capacity But Not Sedation Level Predicts Neural Signatures Of Conscious Processing."
Robin Carhart-Harris, "The Neural Correlates Of Psychedelic Consciousness As Determined By fMRI Studies With Psilocybin."
Concurrent sessions 1.1: Metacognition and higher order consciousness
Tuesday, July 3 2012 14:00-16:00 @ Dome theatre
Concurrent session 1.1: Metacognition and higher order consciousness
Zoltan Dienes, "How To Determine If Knowledge Is Unconscious."
Manuel Rausch, "ERP Correlates Of Consciousness Depend On The Measurement: Decisional Confidence Vs. Visual Experience."
Ryan Scott, "Knowing If They Know: A Novel Bias-Free Method For Incentivising Accurate Metacognitive Reports."
Adam Barrett, "How Can We Know When We Know We Know? Towards Measuring Metacognition."
Christian Beenfeldt, "Titchener’s “Introspectionism” Contra Contemporary Introspective Approaches In Scientific Psychology."
Kevin Reuter, "There Is No Introspective Attention."
Concurrent session 1.1: Metacognition and higher order consciousness
Zoltan Dienes, "How To Determine If Knowledge Is Unconscious."
Manuel Rausch, "ERP Correlates Of Consciousness Depend On The Measurement: Decisional Confidence Vs. Visual Experience."
Ryan Scott, "Knowing If They Know: A Novel Bias-Free Method For Incentivising Accurate Metacognitive Reports."
Adam Barrett, "How Can We Know When We Know We Know? Towards Measuring Metacognition."
Christian Beenfeldt, "Titchener’s “Introspectionism” Contra Contemporary Introspective Approaches In Scientific Psychology."
Kevin Reuter, "There Is No Introspective Attention."
Symposium 1 Talk 3: Brown
Tuesday, July 3 2012 11:00-13:00 @ Dome Theatre
Symposium 1: "Consciousness fading"
Talk 3: “The neural dynamics of loss and recovery of consciousness under general anesthesia”
Emery Brown (Department of Anesthesia, Harvard Medical School, USA)
SUMMARY
General anesthesia is a drug-induced, reversible condition comprised of five behavioral states: unconsciousness, amnesia (loss of memory), analgesia (loss of pain sensation), akinesia (immobility), and hemodynamic stability with control of the stress response. The mechanisms by which anesthetic drugs induce the state of general anesthesia are considered one of the biggest mysteries of modern medicine. We have been using three experimental paradigms to study general anesthesia-induced loss of consciousness in humans: combined fMRI/EEG recordings, high-density EEG recordings and intracranial recordings. These studies are allowing us to establish precise neurophysiological, neuroanatomical and behavioral correlates of unconsciousness under general anesthesia. Combined with our mathematical modeling work on how anesthetics act on neural circuits to produce the state of general anesthesia we are able to offer specific hypotheses as to how changes in level of activity in specific circuits lead to the unconscious state. We will discuss the relation between our findings and two other important altered states of arousal: sleep and coma. Our findings suggest that the state of general anesthesia is not as mysterious as currently believed.
Symposium 1: "Consciousness fading"
Talk 3: “The neural dynamics of loss and recovery of consciousness under general anesthesia”
Emery Brown (Department of Anesthesia, Harvard Medical School, USA)
SUMMARY
General anesthesia is a drug-induced, reversible condition comprised of five behavioral states: unconsciousness, amnesia (loss of memory), analgesia (loss of pain sensation), akinesia (immobility), and hemodynamic stability with control of the stress response. The mechanisms by which anesthetic drugs induce the state of general anesthesia are considered one of the biggest mysteries of modern medicine. We have been using three experimental paradigms to study general anesthesia-induced loss of consciousness in humans: combined fMRI/EEG recordings, high-density EEG recordings and intracranial recordings. These studies are allowing us to establish precise neurophysiological, neuroanatomical and behavioral correlates of unconsciousness under general anesthesia. Combined with our mathematical modeling work on how anesthetics act on neural circuits to produce the state of general anesthesia we are able to offer specific hypotheses as to how changes in level of activity in specific circuits lead to the unconscious state. We will discuss the relation between our findings and two other important altered states of arousal: sleep and coma. Our findings suggest that the state of general anesthesia is not as mysterious as currently believed.
Symposium 1 Talk 2: Boly
Tuesday, July 3 2012 11:00-13:00 @ Dome Theatre
Symposium 1: "Consciousness fading"
Talk 2: “Is propofol-induced loss of consciousness a sleep-like state?”
Melanie Boly (Coma Science Group, University of Liege, Belgium)
SUMMARY
Mechanisms of propofol-induced fading of consciousness, as well as the relationship between propofol anesthesia and sleep remains poorly understood. We will review recent neuroimaging studies investigating neural correlates of loss of consciousness during propofol sedation and non-REM sleep. We will then highlight some commonalities as well as observed differences in terms of spontaneous EEG, or functional and effective connectivity (using high-density EEG, functional MRI or TMS-EEG) between propofol and non-REM sleep. Finally, we will discuss the implications of these results for neural correlates of consciousness and for their use as clinical tools to detect awareness during anesthesia, and in other altered consciousness states.
Symposium 1: "Consciousness fading"
Talk 2: “Is propofol-induced loss of consciousness a sleep-like state?”
Melanie Boly (Coma Science Group, University of Liege, Belgium)
SUMMARY
Mechanisms of propofol-induced fading of consciousness, as well as the relationship between propofol anesthesia and sleep remains poorly understood. We will review recent neuroimaging studies investigating neural correlates of loss of consciousness during propofol sedation and non-REM sleep. We will then highlight some commonalities as well as observed differences in terms of spontaneous EEG, or functional and effective connectivity (using high-density EEG, functional MRI or TMS-EEG) between propofol and non-REM sleep. Finally, we will discuss the implications of these results for neural correlates of consciousness and for their use as clinical tools to detect awareness during anesthesia, and in other altered consciousness states.
Symposium 1 Talk 1: Supp
Tuesday, July 3 2012 11:00-13:00 @ Dome Theatre
Symposium 1: "Consciousness fading"
Talk 1: "Block of intracortical communication by propofol-induced neural hypersynchy"
Gernot Supp (Dept. of Neurophysiology and Pathophysiology University Medical Center, Hamburg, Germany)
SUMMARY
Manipulating consciousness by anesthetic agents is everyday clinical practice. However, the key mechanisms underlying pharmacologically induced breakdown of consciousness are still largely unresolved. Unraveling the neural determinants of loss of consciousness, apart from its obvious medical advances, may shed new light on brain processes relevant for the emergence of consciousness. The presentations of this symposium will discuss complementary findings that suggest convergent conclusions and highlight the implications of anesthesia research for understanding the neural mechanisms of consciousness. The first and the second presentation will focus on propofol anesthesia as a model of drug-induced loss of consciousness, and discuss recent experimental evidence suggesting that propofol anesthesia induces hypersynchronous ongoing activity leading to a brain state during which information processing is severely compromised, and functional integration across different areas is strongly reduced. The second presentation will also highlight potential similarities between the neurophysiological changes induced by propofol and those observed during sleep. The third presentation of the symposium will, in addition to discussing recent modeling work on the mechanisms of propofol anesthesia, provide a broader framework for understanding changes in neural dynamics under general anesthesia.
Symposium 1: "Consciousness fading"
Talk 1: "Block of intracortical communication by propofol-induced neural hypersynchy"
Gernot Supp (Dept. of Neurophysiology and Pathophysiology University Medical Center, Hamburg, Germany)
SUMMARY
Manipulating consciousness by anesthetic agents is everyday clinical practice. However, the key mechanisms underlying pharmacologically induced breakdown of consciousness are still largely unresolved. Unraveling the neural determinants of loss of consciousness, apart from its obvious medical advances, may shed new light on brain processes relevant for the emergence of consciousness. The presentations of this symposium will discuss complementary findings that suggest convergent conclusions and highlight the implications of anesthesia research for understanding the neural mechanisms of consciousness. The first and the second presentation will focus on propofol anesthesia as a model of drug-induced loss of consciousness, and discuss recent experimental evidence suggesting that propofol anesthesia induces hypersynchronous ongoing activity leading to a brain state during which information processing is severely compromised, and functional integration across different areas is strongly reduced. The second presentation will also highlight potential similarities between the neurophysiological changes induced by propofol and those observed during sleep. The third presentation of the symposium will, in addition to discussing recent modeling work on the mechanisms of propofol anesthesia, provide a broader framework for understanding changes in neural dynamics under general anesthesia.
Symposium 1: ch. Engel
Tuesday, July 3 2012 11:00-13:00 @ Dome Theatre
Symposium 1: "Consciousness fading"
Chair: Andreas K. Engel (Dept. of Neurophysiology and Pathophysiology University Medical Center, Hamburg, Germany)
SUMMARY
Introduction by Andreas Engel.
Manipulating consciousness by anesthetic agents is everyday clinical practice. However, the key mechanisms underlying pharmacologically induced breakdown of consciousness are still largely unresolved. Unraveling the neural determinants of loss of consciousness, apart from its obvious medical advances, may shed new light on brain processes relevant for the emergence of consciousness. The presentations of this symposium will discuss complementary findings that suggest convergent conclusions and highlight the implications of anesthesia research for understanding the neural mechanisms of consciousness. The first and the second presentation will focus on propofol anesthesia as a model of drug-induced loss of consciousness, and discuss recent experimental evidence suggesting that propofol anesthesia induces hypersynchronous ongoing activity leading to a brain state during which information processing is severely compromised, and functional integration across different areas is strongly reduced. The second presentation will also highlight potential similarities between the neurophysiological changes induced by propofol and those observed during sleep. The third presentation of the symposium will, in addition to discussing recent modeling work on the mechanisms of propofol anesthesia, provide a broader framework for understanding changes in neural dynamics under general anesthesia.
Symposium 1: "Consciousness fading"
Chair: Andreas K. Engel (Dept. of Neurophysiology and Pathophysiology University Medical Center, Hamburg, Germany)
SUMMARY
Introduction by Andreas Engel.
Manipulating consciousness by anesthetic agents is everyday clinical practice. However, the key mechanisms underlying pharmacologically induced breakdown of consciousness are still largely unresolved. Unraveling the neural determinants of loss of consciousness, apart from its obvious medical advances, may shed new light on brain processes relevant for the emergence of consciousness. The presentations of this symposium will discuss complementary findings that suggest convergent conclusions and highlight the implications of anesthesia research for understanding the neural mechanisms of consciousness. The first and the second presentation will focus on propofol anesthesia as a model of drug-induced loss of consciousness, and discuss recent experimental evidence suggesting that propofol anesthesia induces hypersynchronous ongoing activity leading to a brain state during which information processing is severely compromised, and functional integration across different areas is strongly reduced. The second presentation will also highlight potential similarities between the neurophysiological changes induced by propofol and those observed during sleep. The third presentation of the symposium will, in addition to discussing recent modeling work on the mechanisms of propofol anesthesia, provide a broader framework for understanding changes in neural dynamics under general anesthesia.
Keynote 1: Bayne
Tuesday, July 3 2012 09:15-10:30 @ Dome theatre
Keynote 1: "The unity of consciousness"
Tim Bayne (University of Oxford, Oxford, UK)
SUMMARY
Some theorists maintain that consciousness is necessarily unified; others maintain that although consciousness is typically unified, the unity of consciousness can break down on occasions; and still others argues that the apparent unity of consciousness is an illusion, and that consciousness is typically disunified. This talk provides an overview of the debate between these three approaches to consciousness. I distinguish different forms that the unity of consciousness can take, and I provide some tentative reasons for thinking that there is a sense in which consciousness is necessarily unified. I conclude with a discussion of how debates about the unity of consciousness might inform the development and evaluation of theories of consciousness.
Keynote 1: "The unity of consciousness"
Tim Bayne (University of Oxford, Oxford, UK)
SUMMARY
Some theorists maintain that consciousness is necessarily unified; others maintain that although consciousness is typically unified, the unity of consciousness can break down on occasions; and still others argues that the apparent unity of consciousness is an illusion, and that consciousness is typically disunified. This talk provides an overview of the debate between these three approaches to consciousness. I distinguish different forms that the unity of consciousness can take, and I provide some tentative reasons for thinking that there is a sense in which consciousness is necessarily unified. I conclude with a discussion of how debates about the unity of consciousness might inform the development and evaluation of theories of consciousness.
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